Heart attack is the most frequent death causes in the world. It occurs when the arteries that supply blood to the heart become damaged and blocked by the blood clot. Arteries blockage reduces the supply of oxygen to the heart and leads to the permanent damage of the heart muscle. Another reason for the heart attack can be the sudden and strong spasm of the coronary artery. Spasm abruptly reduces the oxygen intake to the heart and damages it.  Despite the treatment and the healing, part of the muscle turn into the fibrous tissue, the "scar". It cannot contract like the rest of the heart tissue, which leads to the decrease of blood pumping function.

Heart attacks can be prevented by altering the lifestyle and by close monitoring of some biochemical substances in the blood. Maintaining the optimal body weight is crucial, as extra pounds increase the workload for the heart. Regular physical exercises and diet rich in fruits and vegetables and low in sugar is a must for those who want to lower their chances of a heart attack.  However, the weight management program is the most effective, when designed by a professional and based on your individual genetical profile.

Biochemical substances that circulate in our blood provide information on the pathological processes long before the symptoms become evident. Cholesterol is an example of a substance that increases the risk of a heart attack. It contributes to the build up inside the arteries that carry blood to the heart and brain and reduce the oxygen intake, increasing the chances of a heart attack. About 10% of people suffering from an early onset of a heart disease carry mutations in the APOB or PCSK9 genes. Most of the patients with an inherited high cholesterol level need medication to control their cholesterol level, as the lifestyle changes alone do not help.

Higher than average level of amino acid called homocysteine in the bloodstream may indicate the problems in a cardiovascular system. The excess of this amino acid also associated with the risk of type 2 diabetes, stroke, deep vein thrombosis and obesity. Often the reason for the increase in homocysteine production is in the genes. Mutations in MTHFR gene disrupt normal functioning of enzyme Methylenetetrahydrofolate reductase.

Early genetical screening for those mutations and consultation of the certified practitioner can help to set up an action plan to prevent the development of the disease.